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Its central action in research is competitively blocking NNMT's active site, which does two coupled things: it stops the methylation of nicotinamide (a NAD+ precursor) into 1-methylnicotinamide, and it spares S-adenosylmethionine (SAM), the methyl donor the reaction consumes
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[DOI] [PubMed] [Google Scholar] 91.Domes G., Lischke A., Berger C., Grossmann A., Hauenstein K., Heinrichs M., Herpertz S.C
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